Searchable abstracts of presentations at key conferences in endocrinology

ea0050cmw3.5 | Workshop 3: How do I. . . (1) | SFEBES2017

How do I . . . . investigate and manage a patient with Bartter or Gitleman syndrome?

Sayer John

Bartter and Gitelman syndromes are salt wasting alkaloses. These inherited conditions are the result of impairment of sodium chloride reabsorption in the loop of Henle (Bartter) or distal tubule (Gitelman). Secondary hyperaldosteronism occurs as a direct result of renal salt wasting resulting in hypokalaemia and metabolic alkalosis. The tubular defects seen mimic those of long-term loop (Bartter) or thiazide (Gitelman) diuretic use and urinary calcium levels and serum magnesiu...

ea0050cmw3.5 | Workshop 3: How do I. . . (1) | SFEBES2017

How do I . . . . investigate and manage a patient with Bartter or Gitleman syndrome?

Sayer John

Bartter and Gitelman syndromes are salt wasting alkaloses. These inherited conditions are the result of impairment of sodium chloride reabsorption in the loop of Henle (Bartter) or distal tubule (Gitelman). Secondary hyperaldosteronism occurs as a direct result of renal salt wasting resulting in hypokalaemia and metabolic alkalosis. The tubular defects seen mimic those of long-term loop (Bartter) or thiazide (Gitelman) diuretic use and urinary calcium levels and serum magnesiu...